Skin tears and integrity
Skin tears are often an un-diagnosed problem and prevalence as high as 41.5% have been reported in elderly care residents2
A skin tear is a traumatic wound that is caused by a mechanical force. The mechanical force could be shear or friction, or it could be the result of removing an adherent dressing.1
Skin tears can lead to partial or full separation of the skin’s outer layers (epidermis and dermis). They can also lead to a separation of both the epidermis and the dermis from the underlying structures (a full-thickness wound). The severity of a skin tear varies, depending on the depth of the wound. However, skin tears do not extend through the subcutaneous layer of the skin.1
They can occur on any part of the body, but are most common on the hands, arms and legs.1
Our skin is the body’s first line of defence. It acts as a protective barrier, and prevents damage to our internal tissues and organs. However, if our skin becomes frail or fragile, it’s more likely to be damaged. This increases the risk of skin tears, as less force is needed to cause a traumatic injury.2
While skin tears can happen in a variety of patients, the elderly population is particularly at risk. As we age, our skin becomes more fragile. The skin’s ability to heal itself slows down, which can make it even more vulnerable to skin tears.2 (See Figure 1 for more information)
If an elderly patient needs help to carry out daily tasks, such as moving around or washing, this can increase the risk of skin tears. Additionally, elderly patients who have other illnesses, are undergoing certain medical treatments (e.g. chemotherapy) or are on certain medications (e.g. steroids), can have an even higher risk of developing skin tears.1
Figure 1: How our skin changes as we age1
To prevent your patient from getting a skin tear, you can take two steps. One, find out if your patient is at risk. And two, take steps to minimise your patient’s risk. Let’s look at two tools that can help you to do this.
Tool #1: The Skin Tear Risk Assessment Protocol
To find out if your patient is at risk, you can use the Skin Tear Risk Assessment Protocol (See Figure 2).1
This protocol looks at three main risk factors:
- mobility, and
- general health.
Figure 2: Skin Tear Risk Assessment Protocol1
Tool #2: The Risk Reduction Programme Checklist
If you find out that your patient is at risk, use the Risk Reduction Programme Checklist (See Table 2). This will help you protect your patient from skin tears. The checklist gives you a list of tips and suggestions based on the same three factors as the risk assessment: skin, mobility and general health. By controlling the risk factors around your patient, you can help them maintain skin health and avoid injury.1
|Risk factor||What you should do|
The skin and it's function
It is essential to know the structure of the skin to assess and manage wounds.8 It provides a semi-permeable barrier that protects the body from the external environment and maintains homeostasis in the body.19,35
The skin consist of three layers:
- The epidermis - traps moisture19
- The dermis - functionality due to the variety of structures8
- The subcutaneous tissue - insulation and cushions35
How to treat skin tears
This section provides further insight into:
- how to treat skin tears
- how to choose the right skin dressing
The epidermis is the outermost, visible part of the skin.19. It is a highly specialised structure that adapts to environmental conditions.19,28
It provides a physical and chemical barrier to block entry of foreign substances and triggers an immunological response against any pathogens that penetrate the barrier.19,28
It has a thin layer of cells varying in thickness from 0.05 mm on the eyelids to 1.5 mm on the palms and soles.8
Function of the epidermis:19
- Barrier function
- Control of water loss
Protection against UV light, bacteria and allergens
The dermis is the middle layer of the skin and is approximately 15-40 times thicker than the epidermis.4,35
The dermis consist of connective tissue. A semi-fluid substance mainly consisting of collagen and elastin fibres.4,35
The dermis contains:
- Nerve endings
- Blood vessels
- Skin appendages (e.g. hair follicles, sebaceous glands and sweat glands)4,35
The subcutaneous tissue (subcutis) is the lower layer of the skin between the dermis and fascia.35
Subcutaneous fatty tissue can often be visible in the base of deep wounds such as pressure ulcers or traumatic wounds.5
Functions of the subcutis include:35
- Retains moisture
Did you know?
- The skin is the largest organ in the body 10,35
- The skin renews itself every 28 days.36
- The thickest skin is on the feet, at 1,5 mm thick.8
- Every minute 30,000 dead skin cells are shed. 9
The lipids produced in the outer layer of the skin are crucial to the function of the epidermal barrier. The lipid barrier can easily be disrupted by soaps and detergents.19
The average person’s skin when stretched can cover 2 square metres and contain more than 11 miles of blood vessels.34
Skin has its own community of microorganisms called skin flora. Its density and composition varies with anatomical location and can be 1000s per square metre.34
Factors affecting the skin barrier function
Factors influencing the skin barrier function:
- The effects of ageing / Elderly skin
- pH levels of the skin
- Dry skin
- Moisture Associated Skin Damage (MASD)
The effects of ageing
Elderly skin has fewer sebaceous glands resulting in reduced secretion of natural lipids, increasing the need to moisturise.2,29
Elderly skin develops a flattening of the rete ridges that hold the epidermis and dermis together resulting in the higher incidence of skin tears in the elderly.29
Premature babies have fragile, thinner, more immature skin than full term babies, resulting in higher Trans Epidermal Water Loss.19
The skin of full-term babies contains 10-20 layers of stratum corneum. Premature babies only have 2-3 layers.17
The overall result is that preterm babies are more prone to skin trauma from adhesive dressings and tapes and localised pressure from IV lines and tubing.19
The term ‘moisture-associated skin damage’ (MASD) is used to describe all types of skin damage caused by moisture.21
Damage to the skin caused by moisture has a significant impact, decreasing the skin’s ability to act as a functional protective barrier.21
When the skin becomes dry, the superficial corneocytes on the surface attached to the deeper keratinocytes, pile up and become visible as scaly, rough and flaky skin.15
When skin is dry, the function of the acid mantle is reduced resulting in reduced protection against bacteria. This, together with minor trauma from scratching, increases the risk of skin infection.31
Moisture balance is imperative to wound healing
If exudate is not properly managed the periwound skin may become macerated.39
Macerated skin has a higher pH than normal skin and increases the risk of bacterial and fungal infections, such as Candida albicans. 4,19
It is important that excess exudate is removed from the wound by an absorbent dressing with the ability to contain exudate under pressure.1
Chronic wound exudate is rich in: 1
- Growth factors that which promote healing
- Dead white cells, bacteria, high levels of inflammatory mediators and proteases which can disrupt the normal wound healing process
High protease content of chronic wound fluid causes maceration and breaks down of the stratum corneum.19,39
Failure to manage exudate adequately can lead to exposure of the periwound skin to moisture resulting in maceration of the periwound skin.39
ITD is an inflammatory dermatosis occurring as a consequence of moisture, friction and bacteria trapped between two skin surfaces.6
In skin folds, there is a risk that sweat can get trapped leading to build-up of moisture and when the skin rubs together it becomes painful due to skin excoriation. 37
This increases the risk of secondary infection by fungi and bacteria and leads to intertrigo.37
Peri-stomal skin damage occurs when skin is exposed to effluent from an ostomy, resulting in inflammation and erosion.38
Lipases and proteases produced by faecal bacteria break down protein in keratinocytes, contributing to skin breakdown.6
Incontinence Associated Dermatitis (IAD) is a complex mechanism that is not fully understood but involves an interaction between, urine and faeces on the skin, humidity, mechanical irritants and frequent use of soap and water.19
If the skin is exposed to urine, faeces and detergents for a longer time the skin pH becomes more alkaline disturbing the acid mantle and increasing skin damage.19
In the sacral area, even experts have difficulty identifying if skin damage is due to pressure damage or moisture damage.22
An example is patients who are at risk for IAD. They are also at risk for pressure ulcer development, due to impaired tolerance to friction and sheer when IAD is present.6
Stage I pressure ulcers are often mistaken for mild/moderate IAD as both conditions present as erythema of intact skin.6,19
You may also be interested in
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- Beeckman, D., Schoonhoven, L., Fletcher, J., Furtado, K., Gunningberg, L., Heyman, H., Lindholm, C., Paquay, L., Verdú, J. and Defloor, T. (2007). EPUAP classification system for pressure ulcers: European reliability study. Journal of Advanced Nursing, 60(6); 682-691.
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